Journal of immunology (Baltimore, Md. : 1950)
International Standard Serial Number (ISSN)
publication venue for
Complement Receptor 3 Contributes to the Sexual Dimorphism in Neutrophil Killing of Staphylococcus aureus.
Innate Sex Bias of Staphylococcus aureus Skin Infection Is Driven by α-Hemolysin.
Naive T cell repertoire skewing in HLA-A2 individuals by a specialized rearrangement mechanism results in public memory clonotypes.
CD43 regulates Th2 differentiation and inflammation.
Constitutive ERK MAPK activity regulates macrophage ATP production and mitochondrial integrity.
Functional CD8+ T cell responses in lethal Ebola virus infection.
Kinetic proofreading of ligand-FcepsilonRI interactions may persist beyond LAT phosphorylation.
Regulation of N-formyl peptide receptor signaling and trafficking by individual carboxyl-terminal serine and threonine residues.
Increased expression of genes linked to FcepsilonRI Signaling and to cytokine and chemokine production in Lyn-deficient mast cells.
Synergy between extracellular group IIA phospholipase A2 and phagocyte NADPH oxidase in digestion of phospholipids of Staphylococcus aureus ingested by human neutrophils.
Chemotaxis inhibitory protein of Staphylococcus aureus binds specifically to the C5a and formylated peptide receptor.
Fibrinogen depletion attenuates Staphyloccocus aureus infection by preventing density-dependent virulence gene up-regulation.
Regulation of N-formyl peptide-mediated degranulation by receptor phosphorylation.
Expression of P-selectin at low site density promotes selective attachment of eosinophils over neutrophils.
Investigation of neutrophil signal transduction using a specific inhibitor of phosphatidylinositol 3-kinase.
Signal transducing properties of the N-formyl peptide receptor expressed in undifferentiated HL60 cells.
The rabbit neutrophil N-formyl peptide receptor. cDNA cloning, expression, and structure/function implications.