American journal of physiology. Heart and circulatory physiology
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Reduced membrane cholesterol limits pulmonary endothelial Ca(2+) entry after chronic hypoxia.
NFAT regulation of cystathionine γ-lyase expression in endothelial cells is impaired in rats exposed to intermittent hypoxia.
Hydrogen sulfide-induced vasodilation mediated by endothelial TRPV4 channels.
Intermittent Hypoxia in Rats Reduces Activation of Ca2+ Sparks in Mesenteric Arteries.
Endothelin-1-induced vasoconstriction does not require intracellular Ca²⁺ waves in arteries from rats exposed to intermittent hypoxia.
Role of ASIC1 in the development of chronic hypoxia-induced pulmonary hypertension.
Hydrogen sulfide dilates rat mesenteric arteries by activating endothelial large-conductance Ca²⁺-activated K⁺ channels and smooth muscle Ca²⁺ sparks.
Altered membrane lipid domains limit pulmonary endothelial calcium entry following chronic hypoxia.
NFATc3 contributes to intermittent hypoxia-induced arterial remodeling in mice.
Mechanisms of ATP-mediated vasodilation in humans: modest role for nitric oxide and vasodilating prostaglandins.