Alcoholism, clinical and experimental research
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Alcohol Use During Pregnancy is Associated with Specific Alterations in MicroRNA Levels in Maternal Serum.
12-step approaches for the dually diagnosed: mechanisms of change.
Infant stress reactivity and prenatal alcohol exposure.
Moderate fetal alcohol exposure impairs the neurogenic response to an enriched environment in adult mice.
Chronic ethanol consumption reduces delta-and mu-opioid receptor-stimulated G-protein coupling in rat brain.
Fetal alcohol exposure alters GAP-43 phosphorylation and protein kinase C responses to contextual fear conditioning in the hippocampus of adult rat offspring.
Prenatal ethanol exposure reduces mGluR5 receptor number and function in the dentate gyrus of adult offspring.
Chronic prenatal ethanol exposure alters ionotropic glutamate receptor subunit protein levels in the adult guinea pig cerebral cortex.
A mouse model of prenatal ethanol exposure using a voluntary drinking paradigm.
Neurosteroid modulation of glutamate release in hippocampal neurons: lack of an effect of a chronic prenatal ethanol exposure paradigm.
Dose-dependent effects of prenatal ethanol exposure on synaptic plasticity and learning in mature offspring.
Effects of prenatal ethanol exposure on phospholipase C-beta 1 and phospholipase A2 in hippocampus and medial frontal cortex of adult rat offspring.
Brain region-dependent sensitivity of GABAA receptor-mediated responses to modulation by ethanol.
Ethanol-induced changes in chloride flux are mediated by both GABA(A) and GABA(B) receptors.
gamma-Aminobutyric acid-activated chloride channels in rats selectively bred for differential acute sensitivity to alcohol.
Sensitivity to ethanol hypnosis and modulation of chloride channels does not cosegregate with pentobarbital sensitivity in HS mice.
Individual differences in behavioral and subjective responses to alcohol.
Course of recovery from alcoholism.