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Bragin, Denis Associate Professor

Positions

Associate Professor, Department of Neurosurgery

Bragin received his MSc in biochemistry from Rostov State University (Russia) in 1999. He completed his post-graduate work at the Kogan’s Research Institute for Neurocybernetics, where he studied the effects of photodynamic treatment on neurons and defended his PhD in biophysics in Voronezh State University/Russian Academy of Sciences in 2004. He joined UNM’s Department of Neurosurgery in 2005 as a postdoctoral fellow researching epilepsy, stroke and brain trauma. In 2011 he was promoted as an assistant professor at the Department of Neurosurgery.

Research Interests

Bragin′s research focuses on the circulation and metabolism of the brain after the stroke and traumatic brain injury. His current research interests include:

The role of cerebral microvascular shunting in the development of hyperemia, brain edema, tissue ischemia and blood-brain barrier degradation during intracranial hypertension after severe cerebral insults.
The potential of hemodynamics modulation with drag-reducing molecules for treating acute and chronic cerebral microvascular disorders and ischemia including stroke, traumatic injury and neurodegenerative diseases.
Non-invasive transcranial direct current and pulsed electromagnetic field brain stimulations for recovery after acute brain injuries.

Publications
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selected publications

academic article
- Leukemia-derived exosomes and cytokines pave the way for entry into the brain.. Journal of leukocyte biology. 105:741-753. 2019
- Myeloid adrenergic signaling via CaMKII forms a feedforward loop of catecholamine biosynthesis.. Journal of molecular cell biology. 9:422-434. 2017
- Rheological effects of drag-reducing polymers improve cerebral blood flow and oxygenation after traumatic brain injury in rats.. Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism. 37:762-775. 2017
- High Intracranial Pressure Induced Injury in the Healthy Rat Brain.. Critical care medicine. 44:e633-e638. 2016
- In Vivo Inhibition of miR-155 Promotes Recovery after Experimental Mouse Stroke.. The Journal of neuroscience : the official journal of the Society for Neuroscience. 35:12446-12464. 2015
- Enhancement of neurogenesis and memory by a neurotrophic peptide in mild to moderate traumatic brain injury.. Neurosurgery. 76:201-14; discussion 214-5. 2015
- Critical cerebral perfusion pressure at high intracranial pressure measured by induced cerebrovascular and intracranial pressure reactivity.. Critical care medicine. 42:2582-2590. 2014
- Role of microvascular shunts in the loss of cerebral blood flow autoregulation.. Advances in experimental medicine and biology. 812:43-49. 2014
- Effect of cerebral perfusion pressure on cerebral cortical microvascular shunting at high intracranial pressure in rats.. Stroke; a journal of cerebral circulation. 44:177-181. 2013
- Microvascular shunts in the pathogenesis of high intracranial pressure.. Acta neurochirurgica. Supplement. 118:205-209. 2013
- “Microvascular shunts in the pathogenesis of high intracranial pressure” . Acta Neurochir Suppl. 118:205-209. 2013
- Differential changes of glutathione levels in astrocytes and neurons in ischemic brains by two-photon imaging.. Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism. 30:734-738. 2010
- Development of epileptiform excitability in the deep entorhinal cortex after status epilepticus.. The European journal of neuroscience. 30:611-624. 2009
- Critical cerebral perfusion pressure at high intracranial pressure measured by induced cerebrovascular and intracranial pressure reactivity.
- Effect of cerebral perfusion pressure on cerebral cortical microvascular shunting at high intracranial pressure in rats.
- Enhancement of neurogenesis and memory by a neurotrophic Peptide in mild to moderate traumatic brain injury.
- High intracranial pressure effects on cerebral cortical microvascular flow in rats.
- Increases in microvascular perfusion and tissue oxygenation via pulsed electromagnetic fields in the healthy rat brain.
- Photodynamic effect of hypericin and a water-soluble derivative on isolated crayfish neuron and surrounding glial cells.
- Photodynamic inactivation of isolated crayfish mechanoreceptor neuron: different death modes under different photosensitizer concentrations.
- Photodynamic inactivation of isolated crayfish neuron requires protein kinase C, PI 3-kinase and Ca2+.
- nvolvement of adenylate cyclase and tyrosine kinase signaling pathways in response of crayfish stretch receptor neuron and satellite glia cell to photodynamic treatment.