academic article

• Genome-wide association study identifies germline polymorphisms associated with relapse of childhood acute lymphoblastic leukemia..  Blood.  120:4197-4204. 2012
• Outcome modeling with CRLF2, IKZF1, JAK, and minimal residual disease in pediatric acute lymphoblastic leukemia: a Children's Oncology Group study..  Blood.  119:3512-3522. 2012
• Ancestry and pharmacogenomics of relapse in acute lymphoblastic leukemia..  Nature genetics.  43:237-241. 2011
• Key pathways are frequently mutated in high-risk childhood acute lymphoblastic leukemia: a report from the Children's Oncology Group..  Blood.  118:3080-3087. 2011
• Gene expression classifiers for relapse-free survival and minimal residual disease improve risk classification and outcome prediction in pediatric B-precursor acute lymphoblastic leukemia..  Blood.  115:1394-1405. 2010
• Identification of novel cluster groups in pediatric high-risk B-precursor acute lymphoblastic leukemia with gene expression profiling: correlation with genome-wide DNA copy number alterations, clinical characteristics, and outcome..  Blood.  116:4874-4884. 2010
• Rearrangement of CRLF2 is associated with mutation of JAK kinases, alteration of IKZF1, Hispanic/Latino ethnicity, and a poor outcome in pediatric B-progenitor acute lymphoblastic leukemia..  Blood.  115:5312-5321. 2010
• Deletion of IKZF1 and prognosis in acute lymphoblastic leukemia..  The New England journal of medicine.  360:470-480. 2009
• Genome-wide interrogation of germline genetic variation associated with treatment response in childhood acute lymphoblastic leukemia..  JAMA.  301:393-403. 2009